A Post about Coronaviruses

Something has been bothering me for a while, which means this post has been brewing for a while. (It’s also been edited a few times, since posting. Mostly in this introductory section, where I’ve cut and rearranged because the first version had too many words and asides.)

I’m going to talk about coronaviruses.

Marie and Duchess sitting on a carpeted floor, looking up at the camera. Marie is a pale gray watermarked tabby cat. Duchess is a pale gray and orange tortoiseshell cat with a clipped left ear.

This post is a departure for me. In talking/writing about coronaviruses on this blog, I’m breaching the barrier between my two worlds. Between my relaxing world of creativity, where I indulge myself with poetry and photos and blog posts, and my anxious world of responsibilities, where I worry about knowledge and knowledge gaps and the idea that facts about the natural world exist but are seldom fully grasped.

In my anxious world of responsibilities, I’ve been talking about coronaviruses for much of the past year. But only with friends and family. And cats. Marie and Dutch are attentive listeners.


Marie, a pale gray watermarked tabby cat, is looking out of the window from her perch on a flannel blanket at the foot of a bed. Duchess, a pale gray and orange tortoiseshell cat, is sprawled in her back, yawning, in a chair in front of the window.

Actually, they’re not very good listeners at all.

Two cats, Marie and Duchess, are sitting on a carpeted floor, looking into their wire and canvas expandable play tunnels. One tunnel is green, the other is blue. Their heads and shoulders are fully inside the tunnels, so only their backs and tails are visible.

Moving on…

Two cats, Marie and Duchess, are sitting on a red mat, backs to the camera, looking out of a door that has windowpanes extending almost to the floor.

In this post, I’m going to explain some of what I know about feline coronavirus. Then I’m going to explain why I’ve been talking to my friends and family and cats about feline coronavirus during the COVID-19 pandemic.

Part I

The coronavirus family tree

To be clear, feline coronavirus is a distinctly different virus from COVID-19.

Taxonomically, both feline coronavirus and COVID-19 belong to the subfamily orthocoronavirinae (previously called coronavirinae), but they are in different genera. COVID-19 is in the genus betacoronavirus, while feline coronavirus is in the genus alphacoronavirus.

But what do these classifications mean? Obviously, they mean that feline coronavirus and COVID-19 are somewhat related, but does “somewhat related” mean anything useful for bloggers and readers and cats?

Two cats, Marie and Duchess, are looking at the camera while sitting on a gray desk chair that is in front of a white desk. The desk is stacked with papers and a spiral notebook. Marie is a pale gray watermarked tabby cat. Duchess is a pale gray and orange tortoiseshell cat with a clipped left ear.

A linguist might say It depends on what you mean by ‘useful.’ An editor might say The question needs editing before it can be answerable. And a taxonomist would likely say Please stop before you even start, because viral taxonomy follows its own rules and should not be compared to cats.

Two cats, Marie and Duchess, are sitting on a gray blanket with their backs to the camera, looking out of a window. Both have their front feet on the window sill. The tension in their bodies indicates that they are very interested in whatever is off-camera, outside the window.

The history of the taxonomy, classification, and nomenclature of viruses is an interesting study of its own. Efforts to classify viruses began in the 1960s and continue today, with a major expansion of the classification system having been proposed as recently as 2017. Currently, the family tree of coronaviruses looks something like this (Decaro & Lorusso 2020; Kipar & Meli, 2014):

  • Order – Nidovirales
    • Family – coronaviridae
      • Subfamily – coronavirinae (as of 2014) or orthocoronavirinae (as of 2020)
        • Genus – alphacoronavirus, betacoronavirus, deltacoronavirus, and gammacoronavirus

Beyond the genus level of classification, the coronavirus family tree branches into subgenera, species, and subspecies, with some 39 species of coronaviruses distributed across 27 subgenera (Coronaviridae Study Group of the International Committee on Taxonomy of Viruses, 2020).

But again, what do these classifications mean?

In a desperate and thoroughly unscientific attempt to answer this question, I’m borrowing an example from mammalian taxonomy. (Remember the taxonomist’s warning, that viral taxonomy should not be compared to cats? Like I said, the following comparison is thoroughly unscientific. I’ll understand if the taxonomist, or any other reader, snorts in contempt and walks away.)

Two cats, Marie and Duchess, are sleeping together in a cat bed in front of a window. The cat bed is slightly too small for both of them to fit comfortably, so they look slightly uncomfortable. Beside them, there is a second cat bed, which is empty.

In viral taxonomy, feline coronavirus and COVID-19 are in the same subfamily, but in different genera.

In mammalian taxonomy, domestic cats and bobcats are in the same subfamily, but in different genera.

The feline family tree

Dutch and Marie are domestic cats. Spoiled, pampered, much loved house cats.

Two cats, Marie and Duchess, are curled up together on a brightly colored blanket on the floor in front of a glass door. Their backs are to the camera, and they are looking outside.

Taxonomically speaking, Dutch and Marie belong in subfamily Felinae, the genus Felis, and the species catus. Put in the more familiar binomial phrasing of genus-species, Dutch and Marie are Felis catus. By comparison, bobcats also belong to the subfamily Felinae, but are classified in the genus Lynx and species rufus. So, binomially, bobcats are Lynx rufus.

To add a third, and somewhat more complicated, data point (because everything is complicated in taxonomy), Pallas’s cats are also classified in the subfamily Felinae. But some sources place Pallas’s cats in the genus Felis and other sources separate them into the genus Otocolobus. All seem to agree on a species name for Pallas’s cats–manul. So Pallas’s cats are variously listed as Felis manulOtocolobus manul, or Felis (Otocolobus) manul.

Marie and Dutch, being pair-bonded rescue Felis catus, are clearly related to each other. Littermates, maybe. But they are only distantly related to bobcats and Pallas’s cats. Some taxonomists, those who classify Pallas’s cats in the genus Felis, might consider Marie and Dutch more closely related to Pallas’s cats than they are to bobcats. Other taxonomists, those who classify Pallas’s cats in the genus Otocolobus, might consider Marie and Dutch no more closely related to Pallas’s cats than they are to bobcats. For my purposes, it is enough to note that domestic cats, bobcats, and Pallas’s cats are all cats, but they are all distinctly different cats.

A gray cat, Duchess, is mostly hidden in a brown polka-dot cat bed with tall sides. All that can be seen over the sides of the bed are her ears and one hind foot extended over the bed's side, toward the camera. The pads of her foot are visible.

Feline coronavirus and COVID-19 are both coronaviruses, but they are distinctly different coronaviruses.

(Back to the taxonomist’s concerns: Viral taxonomy and mammalian taxonomy are, indeed, different systems. The above comparison is flagrantly unscientific. I offer it as a metaphorical demonstration of the messiness inherent in trying to describe, measure, or quantify relatedness among viruses and/or cats.)

A gray cat, Marie, is mostly hidden under a black sheet and white blanket. All that can be seen is her nose and part of one eye, along with a few toes of one front foot. Her position and tension indicate that she is stalking something from her hiding place under the sheet and blanket.

Part II

As recently as the early 1990s, when I first entered veterinary school, there were many knowledge gaps in the story of feline coronavirus. Now research has illuminated how the virus moves within cat populations and has unraveled some of the complex mechanisms that mediate how the virus affects individual cats.

From here, for the sake of brevity and clarity, I’m going to shorten “feline coronavirus” to FCoV. For one thing, I won’t have to keep typing the whole name. For another, I want to be as clear as possible that coronaviruses are a large and varied group of viruses, while FCoV is a very specific coronavirus that infects cats. Probably even Marie and Dutch, at some point in their lives.

Two cats, Marie and Duchess, are looking at the camera from their positions curled up together in a gray checked cat bed. They were asleep together and just woke up. Their expressions indicate they are interested in the camera.

Yes, even you, my dears. But it’s okay, because the overwhelming majority of cats that become infected with FCoV will have few or no symptoms. Perhaps some diarrhea or other gastrointestinal signs, perhaps some upper respiratory congestion.

(There’s more to the FCoV story, which I’ll come to later. For now, I’ll simply say that I’m grateful Marie and Dutch are among the overwhelming majority of cats who have avoided the “more” part of the FCoV story.)

Two pale gray cats, Marie and Duchess, are sleeping facing each other. Duchess has one front foot wrapped over Marie's neck, and Marie has both front feet propped on Duchess's chest. They are in front of a window, and grass is visible in the background.

Marie and Dutch have likely been infected with FCoV, perhaps on multiple occasions. Because FCoV is “worldwide and ubiquitous among virtually all cat populations”, found in more than 60% of pet cats in multi-cat households and in as many as 90% of kittens in shelters (Pedersen, 2009, p. 227).

FCoV is a single-stranded RNA virus

The particular feature of FCoV that is important to this post, and that has been important in my year-long discussions with friends and family, relates to the way coronaviruses carry their genetic information. Unlike humans and cats (and most other organisms), who carry their genetic information as double strands of DNA, coronaviruses carry their genetic information as single strands of RNA. So FCoV, like all other coronaviruses, employs single-stranded RNA as the primary molecule for carrying genetic information.

Dutch and Marie always go to sleep at this point. It’s okay if you do, too. I’ve fallen asleep several times, myself. But there is a point to this post. I’m getting close to it, and my next tangent about the differences between double-stranded DNA and single-stranded RNA will get even closer.

Two gray cats, Marie and Duchess, are sleeping nose-to-nose curled up together on blanket. The camera is very close to their heads, so their ears are prominently visible.

The double helix packaging of DNA provides a relatively stable structure for passing along genetic information. Each strand of DNA serves as a sort of back-up copy for its partner strand, and the process of DNA copying actually uses this back-up feature to proofread and correct mistakes. Should a strand of DNA break, or should mistakes occur in copying a strand, the back-up copy allows enzymes to repair the breaks and remedy the mistakes. This prevents mutations. Obviously, some mutations slip through, but at a far lower rate than would otherwise occur.

Single strands of RNA are less stable genetic carriers than double-stranded DNA. RNA is a more fragile molecule than DNA, and single-stranded RNA, lacking partner strands, has no back-up copies for enzymatic proofreading. Coronaviruses do have a unique mechanism for proofreading, a complex of enzymes and proteins that proofread key genes (Robson et al., 2020), and this unique mechanism provides some stability. But rapid and frequent mutations still occur.

As a single-stranded RNA virus, FCoV does a poor job of creating exact copies of itself. Every time FCoV copies itself, errors occur. Every time (Kipar & Meli, 2014, p. 507). For that matter, FCoV mutates so often that researchers characterize the array of viruses produced in the course of a single infection as a quasispecies–a group of “related genotypes” (Kipar & Meli, 2014, p. 507). Other researchers use the term “pseudo-strain” (Emmler et al., 2020, p. 792).

In short, within any FCoV infected cat, there are many mutated versions of the FCoV they originally contracted.

Two gray cats are snuggled together in front of a window. The camera is very close. Marie is sleeping with her forehead pressed against Duchess's back. Marie's nose and whiskers are visible. Duchess has her back to the camera and has fallen asleep while looking out of the window.

FCoV and feline infectious peritonitis

The overwhelming majority of cats that become infected with FCoV will have few or no symptoms. Perhaps some diarrhea or other gastrointestinal signs, perhaps some upper respiratory congestion. But there’s more to the story.

For somewhere between about 1% (Pedersen et al., 2012, p. 20) and 12% (Addie et al., 2009, p. 594) of infected cats, their particular FCoV quasispecies mutates into one of a number of forms that are able to cause a devastating and often fatal disease: feline infectious peritonitis (FIP).

I say “able to cause” because the FIP-able quasispecies do not always cause FIP. Some cats can resist FIP, even when their FCoV infection mutates into a form capable of causing FIP. Some cats are resistant at one point in their lives and later become susceptible, others perhaps follow an opposite path. In essence, FIP occurs at intersections between rapidly mutating FCoV quasispecies and the genetics and immune systems of individual cats. When an FCoV quasispecies gains the ability to cause FIP, in a cat that never had or has lost the ability to resist FIP, a deadly cascade of disease may begin.

What I’ve just described is the internal mutation theory of FIP. Put bluntly, this theory says that every case (or cluster of cases) of FIP represents a newly mutated variant of FCoV that is newly capable of causing FIP.

And now, all tangents complete, I come to the point of this post.

Two gray cats, Marie and Duchess, are stretched out together on a blue tie-dyed blanket in front of a window. Marie is asleep with her front feet extended, and Duchess is almost asleep, holding her head up, still, but her eyes are closed.

FIP is not rare.

  • As of 2008, FIP was “one of the leading infectious causes of death among young cats from shelters and catteries” (Pedersen, 2009, p. 225).
  • “In one study, FIP was the most common single cause of disease in cats younger than 2 years of age…. An average of 1-5% of young cattery or shelter cats in the US will die from FIP, with losses in catteries higher than from shelters” (Pedersen, 2009, p. 227).
  • “Up to 12% of FCoV-infected cats may succumb to FIP, with stress predisposing to the development of disease” (Addie et al., 2009, p. 594).

This is the source of my bother. (Remember the bother, all those paragraphs ago, that started this post?)

What does it all mean?

I haven’t found much information about the mutation rates of COVID-19. I feel like the data exists, at least in some rough estimate, but I’ve not found it in a reliable and readily accessible format. And, without ready access to the mutation rates of COVID-19, my frame of reference reverts to my existing knowledge about FCoV.

Two cats are silhouetted in front of a screened window. Both cats have their backs to the camera and are looking with interest at something outside the window. Grass is visible outside.

FCoV and COVID-19 are only distantly related, but all coronaviruses share the genetic instability that comes from having a single-stranded RNA genome. Yes, coronaviruses have a unique mechanism for some stability, but this mechanism can’t completely compensate for the instability that leads to mutations.

A vague measure of the instability of FCoV can be seen in the incidence of FIP in cats around the world. Because each case (or cluster of cases) of FIP represents an FCoV quasispecies that has newly acquired one or more of the mutations that enable FIP.

Remember those word problems in math class?

  1. Think about how many kittens and young cats there are in the world. (While no one counts the number of kittens born each year, the ASPCA estimates that 3.2 million cats enter US animal shelters each year…)
  2. Narrow the number down to all the kittens and young cats in shelters and multi-cat environments, each year. (Hint: That’s still so very many cats.)
  3. Between 60% and 90% of the cats in shelters or in multi-cat environments will, at some point, become infected with FCoV.
  4. Calculate a number that would be between 1% and 12% of FCoV-infected kittens and young cats.

That’s how many cats will develop FIP each year.

According to the internal mutation theory, that’s how many times FCoV mutates, each year, into a form capable of causing FIP in a cat that is incapable of resisting FIP. (To determine the exact number of times FCoV mutates into a form capable of causing FIP, add the times such mutations occur in a resistant cat.)

As a word problem, the math itself is not too complicated. The scope of the problem is obvious, even without exact numbers.

Limiting the emergence of variants is the point

The incidence of FIP represents a direct measure of how often one specific group of FCoV variants emerge in cats. Each case (or cluster of cases) of FIP represents a newly mutated variant of FCoV that is newly capable of causing FIP. And FIP is not rare.

I’ve spent the last year lecturing my family and friends and cats about the mutation rate of FCoV, pleading for everyone to do as much as possible to limit COVID-19’s infection cycles.

Yes, it’s true that FCoV and COVID-19 are only distantly related. (Metaphorically, about as distantly related as domestic cats are to bobcats.) But if the mutation rate of COVID-19 is even a fraction of what is seen with FCoV, the risk of new variants surges with each surge of infections.

While it is scientifically inaccurate and somewhat irresponsible to claim that more dangerous COVID-19 variants are inevitable if infections continue, it is equally inaccurate and irresponsible to claim that more dangerous variants are impossible. This, also, is the point.

A slightly overweight cat (sorry, Duchess, but it's true) is standing on her hind legs with her front feet braced against a closed window. She is silhouetted, and grass, shrubbery, and a wooden fence are visible outside.

P.S. Marie and Duchess (Dutch) would like me to add that they are very good listeners, all the time. It’s just that they prefer listening to things other than my voice.


Addie, D., Belák, S., Boucraut-Baralon, C. Egberink, H., Frymus, T., Gruffydd-Jones, T., Hartmann, K., Hosie, M. J., Lloret, A., Lutz, H., Marsilio, F., Pennisi, M. G., Radford, A. D., Thiry, E., Truyen, U., & Horzinek, M. C. (2009). Feline infectious peritonitis: ABCD guidelines on prevention and management. Journal of Feline Medicine and Surgery 11, 594-604. doi: 10.1016/j.jfms.2009.05.08

Coronaviridae Study Group of the International Committee on Taxonomy of Viruses (2020). The species Severe acute respiratory syndrome-related coronavirus: classifying 2019-nCoV and naming it SARS-CoV-2. Nature Microbiology 5, 536-544. https://doi.org/10.1038/s41564-020-0695-z

Decaro, N. & Lorusso, A. (2020). Novel human coronaviruses (SARS-CoV-2): A lesson from animal coronaviruses. Veterinary Microbiology 244(2020). 1-18. doi: 10.1016/j.vetmic.2020.108693

Emmler, L., Felten, S., Matiasek, K., Balzer, H.-J., Pantchev, N., Leutenegger, C., & Hartmann, K. (2020) Feline coronavirus with and without spike gene mutations detected by real-time RT-PCRs in cats with feline infectious peritonitis. Journal of Feline Medicine and Surgery 22(8). 791-799. doi: 10.1177/1098612X19886671

Kipar, A. & Meli, M. L. (2014). Feline infectious peritonitis: Still an enigma? Veterinary Pathology 51(2). 505-526. doi: 10.1177/0300985814522077

Pedersen, N. C. (2009). A review of feline infectious peritonitis virus infection: 1963-2008. Journal of Feline Medicine and Surgery 11. 225-258. doi: 10.1016/j.jfms.2008.09.008.

Pedersen, N. C., Liu, H., Scarlett, J., Leutenegger, C. M., Golovko, L., Kennedy, H., & Kamal, F. M. (2012). Feline infectious peritonitis: Role of the feline coronavirus 3c gene in intestinal tropism and pathogenicity based upon isolates from resident and adopted shelter cats. Virus Research 165, 17-28. doi: 10.1016/j.virusres.2011.12.020

Robson, F., Khan, K. S., Le, T. K., Paris, C., Demirbag, S., Barfuss, P., Rocchi, P., & Ng, W.-L. (2020). Coronavirus RNA proofreading: Molecular basis and therapeutic targeting. Molecular Cell 79, 710-727. https://doi.org/10.1016/j.molcel.2020.07.027

The Cat Eulogies

Vanna (1999-2016)

When we lost Scamper last spring, we were already in the process of losing Vanna, too. Vanna had been diagnosed with intestinal lymphoma two years earlier, and, after thriving for longer than expected under the excellent care of her veterinarian, she was beginning to lose ground.

Vanna had been my mother’s cat, which undoubtedly contributed to the depth of my attachment. She was a living link to an unrecoverable past.

What’s more, she flourished in Virginia. In Tennessee, among Mother’s four cats, Vanna had been the neurotic one. The reclusive, skittish one, rarely glimpsed by visitors.

In Virginia, she became the dominant personality in our household.

When the cancer finally overwhelmed her, almost exactly a month after we said goodbye to Scamper, I stumbled into another depression.

Our lively household of three cats had been reduced, in a month’s time, to a quiet household of one. I couldn’t write about Vanna’s death. Could barely talk about it.

Within a year we were losing Sabrina, too.

Sabrina (2001-2017)

Sabrina was the sweetest, gentlest cat I’ve ever owned. Perhaps the sweetest and gentlest cat I’ve ever met.

She and Scamper had been rescued, at only a few weeks of age, from a construction site.

She suffered a serious injury at about twelve weeks old, losing one of her eyes and undergoing multiple surgeries to salvage the vision in her other eye. She lived the rest of her life with a slowly advancing cataract, but didn’t seem bothered by her limited vision.

She played and romped through adolescence, survived an episode of liver failure in early middle-age, and settled into her senior years with the same calm serenity she had shown from kittenhood.

I had hoped, of course, that we might have a few more years with her, after losing Scamper and Vanna in such close succession. But in November Sabrina began showing signs of discomfort while defecating, our first hint of the rectal tumor that, while repeatedly testing benign on biopsy, was likely malignant at its core.

By March she was too uncomfortable to continue. So I made yet another last trip to our wonderful vet and said yet another goodbye.

How many goodbyes, now? Four, since starting this blog. Indigo. Scamper. Vanna and Sabrina. Before them, Spice.

Spice (?-2008)

Spice’s years as a feral cat ended in 1994, the moment I saw her huddled in the back of a cage with a vast, scabbed wound covering her neck and shoulders. She nosed forward to sniff my hand, speaking in unmistakable cat-language. My name is Spice, and I’ve been waiting for you.

Spice was my constant companion for fifteen years. We shared a dorm, an apartment, a duplex (with my future husband), and, in her final years, a house in the suburbs.

She taught Sabrina and Scamper how to be cats, and they kept her young longer than time should have allowed.

Losing her closed a door on my twenties and thirties. I would never be twenty or thirty again, and I would never have another cat like Spice.

All those that came before

Before Spice? The list is long, stretching through memory into the hazy nostalgia of childhood. Mischief and Jackson. Diana. Gizmo and Annie. Morgan and Shere Khan. Sadie and Daisy. Sheena and Poppy. (This list is far from complete, and includes none of the dogs. I’ll save dogs for a later post.)

Many of our cats were named for characters in books and movies. Some came to us already named, relinquished by owners who could no longer keep them, owners who were happy to let an eager young vet assistant adopt the cats they were losing to eviction, a family illness, or one of life’s other jarring turns.

Some of the cats materialized out of thin air, simply showing up in the yard. Others were dumped on the driveway, plucked from parking lots, and chased down in ditches by a trio of sisters who found it biologically impossible to just keep driving. Mother simply sighed and made room for them all, a tide of cats drifting in and out of our lives, in and out of the house each morning and night.

They were never all in the house at the same time, thankfully. Most preferred the yard, sheds, and pasture, most of the time.


Cats have been one of the few constants in my life. They’ve shared all of my memories, every place I’ve ever called home, and almost every job I’ve ever had. I don’t know how to be without cats. In the end, loving cats is part of how I love myself. So…

Meet Duchess and Marie

Cat Team 7 is a local rescue group who work primarily with cats living at Naval Station Norfolk. The majority of their mission involves a Trap-Neuter-Relocate program, but they sometimes have adoptable kittens.

Duchess and Marie (two of a group named for the Aristocats) were trapped in a warehouse in early June, along with two male kittens about the same age. I saw their photo on social media, contacted Cat Team 7, and the rest is happy history.

They were quite shy, in their first weeks here.

Duchess (or Dutch, because sometimes she’s more Killjoy than Aristocat)

Marie (just Marie, because it fits)

It didn’t take them long to settle in. They have plenty of windows, soft beds, toys, and treats.

They are closely bonded, more dependent on each other than Sabrina and Scamper were. They’re rarely apart.

(Except when Marie plays fetch. Dutch, who has no interest in fetching, stalks the action until she can tempt Marie into a thunderous, romping game of chase.)

And me? I’m sharing my life with cats again. That’s enough for now.


Recommended reading about topics that are more urgent and more important than my cat memories:

Finally, here are three of my favorite recently-read books. Have you read them? What did you think?

Poetry: Who’s Afraid of Black Indians? by Shonda Buchanan

Fiction: Middlesex by Jeffrey Eugenides

Non-fiction: NeuroTribes: The Legacy of Autism and the Future of Neurodiversity by Steve Silberman (I haven’t finished this one yet, but it’s already one of the best books I’ve ever read)

Scamper, 2001-2016


In 2001 we moved from a small duplex to a house with a garage and yard. We had one cat and one dog, and soon added a pair of orphaned kittens.


One of the kittens (on the left in the above photos) was sweet and gentle, and the name “Sabrina” seemed just right for her. The other kitten was fierce and playful and somewhat neurotic, and she defied naming. Nothing quite fit. She became “Scamper” on her medical chart, because I had to put something on her chart, but at home she changed names as often as she changed moods. She was The Scamperer. Thing 2. Her Neediness. The Bad Cat. The Wee Baby Kitty. Supercat.


She was The Bird Watcher. (When it was too cold for open doors and windows, birds on television were better than no birds at all.)


Cats May 15

Cat March 26

In her middle years, she grew overweight and lazy. None of her kitten names fit anymore, if they ever had. She was still Scamper when she went to the vet, but at home she was Herself.

Scamper 2014

Cats Aug 24

In many ways, she was the essence of what I love about cats — neuroses and all.

Scamper 2008

In her later years she liked blankets and patches of sunlight and, every so often, an afternoon nap on the couch with me.

Scamper Jan 2011

Health problems came with age. There were medicines and special diets, all of which helped for a time, but as 2015 progressed her condition declined steadily. She lost weight faster than medicine, food, and love could counter. The calendar turned and she lost more weight. Then, in early February, she stopped eating altogether.

So I made one last trip to the vet with our cat of many names. I stayed with her through the euthanasia process, which was gentle and peaceful, and drove home to a house that is achingly incomplete. There’s an empty spot near her favorite upstairs window, one that can never be filled.

Cat Nov 9

Note: Most of the photos in this post were taken by my husband.

A Few Steps Closer to Spring

Weeds March 16

I usually count the first open-windows day as the first day of spring, but this year I confused the issue by cheating. One day last week, desperate for fresh air, I opened the windows and wore a coat in the house for a few hours. Which means I can’t count yesterday as the first.

Cat March 10

February is always a tough month for me. Its cold, sun-starved days routinely trigger new bouts of depression and anxiety. March, on the other hand, is usually a month of recovery.

Yard March 16

Yard March 16

And if this year’s recovery has been slower to start and harder to sustain than previous years, it has at least begun.

Hyacinth March 16

Hyacinth March 16

Honeysuckle March 16

Honeysuckle March 16

The weather forecast promises a return of winter before the week is finished, but the lengthening days will not allow it to stay.

Hydrangea March 16

Soon the yard will be overrun and winter will fall away into memory, as it does every year.

Brown Thrasher March 16

Robin March 17

Rabbit March 17

Recovering from a Wintry Week

Window April 22

*Cue Announcer* 

We now return to our regularly scheduled season, already in progress…

Live Oak April 13

Dragonfly April 14


Wax Myrtle April 22


Rabbit April 22

Petunia April 21

Flowers April 2

Honeysuckle April 22

Maple April 21

Bee in Tulip April 22